c16 (pkr inhibitor) Search Results


c16  (Bio-Techne corporation)
93
Bio-Techne corporation c16
C16, supplied by Bio-Techne corporation, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/c16+%28pkr+inhibitor%29/bio-techne+corporation___5382?v=Bio-Techne+corporation
Average 93 stars, based on 1 article reviews
c16 - by Bioz Stars, 2026-06
93/100 stars
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hexadecanoic acid (c16, palmitic acid)  (Merck KGaA)
90
Merck KGaA hexadecanoic acid (c16, palmitic acid)
Hexadecanoic Acid (C16, Palmitic Acid), supplied by Merck KGaA, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/c16+%28pkr+inhibitor%29/pm36430935-346-14-19?v=Merck+KGaA
Average 90 stars, based on 1 article reviews
hexadecanoic acid (c16, palmitic acid) - by Bioz Stars, 2026-06
90/100 stars
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c16 (pkr inhibitor)  (ApexBio)
90
ApexBio c16 (pkr inhibitor)
PERK and GCN2 are involved in eIF2α phosphorylation during DHAV-1 infection. (A) Screen the kinases that affect eIF2α phosphorylation. DEFs were infected with DHAV-1 at MOI of 1. After 22 h of infection, different concentrations of kinase inhibitors were added to DEFs for 2 h. Then, DEFs were harvested for immunoblot analysis with the indicated antibodies. (B) PERK inhibitor GSK2606414 inhibits eIF2α phosphorylation induced by DHAV-1. (C) GCN2 inhibitor GCN2-IN-1 inhibits eIF2α phosphorylation induced by DHAV-1. (D) PKR inhibitor <t>C16</t> cannot inhibit eIF2α phosphorylation induced by DHAV-1. (E) Transfection of poly(I:C) activate PKR kinase. (F) DHAV-1 and poly(I:C) stimulate PKR transcription. Differences between two groups were analyzed using Student’s t -test and considered as significant at * p < 0.05, ** p < 0.01, and *** p < 0.001. The bands marked by asterisk (*) are non-specific proteins.
C16 (Pkr Inhibitor), supplied by ApexBio, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/c16+%28pkr+inhibitor%29/pmc08072014-58-0-10?v=ApexBio
Average 90 stars, based on 1 article reviews
c16 (pkr inhibitor) - by Bioz Stars, 2026-06
90/100 stars
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imidazolo-oxindole pkr inhibitor c16  (Cayman Chemical)
90
Cayman Chemical imidazolo-oxindole pkr inhibitor c16
PERK and GCN2 are involved in eIF2α phosphorylation during DHAV-1 infection. (A) Screen the kinases that affect eIF2α phosphorylation. DEFs were infected with DHAV-1 at MOI of 1. After 22 h of infection, different concentrations of kinase inhibitors were added to DEFs for 2 h. Then, DEFs were harvested for immunoblot analysis with the indicated antibodies. (B) PERK inhibitor GSK2606414 inhibits eIF2α phosphorylation induced by DHAV-1. (C) GCN2 inhibitor GCN2-IN-1 inhibits eIF2α phosphorylation induced by DHAV-1. (D) PKR inhibitor <t>C16</t> cannot inhibit eIF2α phosphorylation induced by DHAV-1. (E) Transfection of poly(I:C) activate PKR kinase. (F) DHAV-1 and poly(I:C) stimulate PKR transcription. Differences between two groups were analyzed using Student’s t -test and considered as significant at * p < 0.05, ** p < 0.01, and *** p < 0.001. The bands marked by asterisk (*) are non-specific proteins.
Imidazolo Oxindole Pkr Inhibitor C16, supplied by Cayman Chemical, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/c16+%28pkr+inhibitor%29/pm30817950-42-1-8?v=Cayman+Chemical
Average 90 stars, based on 1 article reviews
imidazolo-oxindole pkr inhibitor c16 - by Bioz Stars, 2026-06
90/100 stars
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imidazolo-oxindole pkr inhibitor c16  (Toronto Research Chemicals)
N/A
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imidazolo-oxindole pkr inhibitor c16  (Aladdin Scientific Corporation)
N/A
Store at -20°C. Store under desiccating conditions. The product can be stored for up to 12 months.
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Image Search Results


PERK and GCN2 are involved in eIF2α phosphorylation during DHAV-1 infection. (A) Screen the kinases that affect eIF2α phosphorylation. DEFs were infected with DHAV-1 at MOI of 1. After 22 h of infection, different concentrations of kinase inhibitors were added to DEFs for 2 h. Then, DEFs were harvested for immunoblot analysis with the indicated antibodies. (B) PERK inhibitor GSK2606414 inhibits eIF2α phosphorylation induced by DHAV-1. (C) GCN2 inhibitor GCN2-IN-1 inhibits eIF2α phosphorylation induced by DHAV-1. (D) PKR inhibitor C16 cannot inhibit eIF2α phosphorylation induced by DHAV-1. (E) Transfection of poly(I:C) activate PKR kinase. (F) DHAV-1 and poly(I:C) stimulate PKR transcription. Differences between two groups were analyzed using Student’s t -test and considered as significant at * p < 0.05, ** p < 0.01, and *** p < 0.001. The bands marked by asterisk (*) are non-specific proteins.

Journal: Frontiers in Microbiology

Article Title: Duck Hepatitis A Virus Type 1 Induces eIF2α Phosphorylation-Dependent Cellular Translation Shutoff via PERK/GCN2

doi: 10.3389/fmicb.2021.624540

Figure Lengend Snippet: PERK and GCN2 are involved in eIF2α phosphorylation during DHAV-1 infection. (A) Screen the kinases that affect eIF2α phosphorylation. DEFs were infected with DHAV-1 at MOI of 1. After 22 h of infection, different concentrations of kinase inhibitors were added to DEFs for 2 h. Then, DEFs were harvested for immunoblot analysis with the indicated antibodies. (B) PERK inhibitor GSK2606414 inhibits eIF2α phosphorylation induced by DHAV-1. (C) GCN2 inhibitor GCN2-IN-1 inhibits eIF2α phosphorylation induced by DHAV-1. (D) PKR inhibitor C16 cannot inhibit eIF2α phosphorylation induced by DHAV-1. (E) Transfection of poly(I:C) activate PKR kinase. (F) DHAV-1 and poly(I:C) stimulate PKR transcription. Differences between two groups were analyzed using Student’s t -test and considered as significant at * p < 0.05, ** p < 0.01, and *** p < 0.001. The bands marked by asterisk (*) are non-specific proteins.

Article Snippet: C16 (PKR Inhibitor) and GSK2606414 (PERK inhibitor) were purchased from APExBIO, and GCN2-IN-1 (GCN2 inhibitor) was purchased from MCE.

Techniques: Phospho-proteomics, Infection, Western Blot, Transfection